Hearing Loss, Dementia and Alzheimer’s Disease: What the Research Shows
Researchers have repeatedly found that older adults with hearing loss are more likely to experience cognitive decline and dementia. That does not mean hearing loss automatically causes dementia, and hearing aids are not a cure for Alzheimer’s disease. It does mean hearing deserves serious attention as part of healthy aging, accurate communication and respectful dementia care.
Four points families should know
There is a real association
Across multiple long-term studies, hearing loss is associated with a higher likelihood of cognitive decline and dementia.
Association is not certainty
Many people with hearing loss never develop dementia. Dementia has many genetic, vascular, medical and social risk factors.
Treatment evidence is promising but specific
The ACHIEVE randomized trial found no overall cognitive benefit across every participant, but did find slower decline among adults at higher baseline risk.
Communication help matters today
Better hearing access can reduce misunderstanding, listening strain, withdrawal and caregiver frustration—even when cognition does not change.
What this page is really saying
The detailed studies below matter, but families should not have to read every hazard ratio or brain-imaging paper to understand the main point. These six answers provide the roadmap.
Is there a connection?
Yes. Across many long-term studies, people with measured hearing loss have experienced cognitive decline or dementia more often than people with better hearing.
Does hearing loss cause dementia?
That has not been proven. Hearing loss may contribute for some people, share causes with dementia, or sometimes appear alongside early brain changes.
Why might they be connected?
Researchers are studying listening effort, reduced sound reaching the brain, social withdrawal, vascular health, central speech processing and reverse causation.
Do hearing aids prevent Alzheimer’s disease?
No responsible provider can promise that. The strongest trial found no overall benefit for every participant, but did find slower decline in adults at higher cognitive risk.
Why test hearing objectively?
People often underestimate their own hearing loss. Missed words can also look like confusion, poor memory or failure to follow instructions.
What should a family do now?
Address hearing for communication, safety and participation. Include trusted support when appropriate and choose technology the person can realistically manage.
Only have a few minutes? Read this roadmap, the section on Gwen’s dementia-aware care and the final plain-English summary. The research atlas remains available for anyone who wants the full evidence.
The Convergence of Auditory Health and Neurodegeneration
Age-related hearing loss and dementia were once discussed as separate consequences of aging. A growing body of epidemiological, clinical, imaging and intervention research now treats hearing as part of the larger brain-health picture. The relationship is important, but it must be described accurately: hearing loss is associated with cognitive decline and dementia risk; it is not proof that every case of hearing loss causes dementia, and no percentage can predict one person’s future.
More than 55 million
WHO estimated that more than 55 million people were living with dementia in 2021, with projections of 78 million by 2030 and 139 million by 2050.
More than 1.5 billion
WHO reports that more than 1.5 billion people live with some degree of hearing loss and about 430 million require hearing-rehabilitation services.
$1.3 trillion
WHO estimated the worldwide cost of dementia at $1.3 trillion in 2019. Its 2030 projection was $1.7 trillion, or $2.8 trillion when increases in care costs are included.
7% population fraction
The 2024 Lancet Commission estimated that hearing impairment was associated with about 7% of dementia cases at the population level, tied with high LDL cholesterol as the largest single modeled fraction.
Why some articles say 8.2% and newer ones say 7%: The 2020 Lancet Commission estimated an 8.2% population-attributable fraction for midlife hearing loss. The 2024 Commission updated the model, risk estimates and prevalence inputs and reported 7%. These are theoretical population estimates—not the percentage of an individual’s personal risk and not proof that eliminating hearing loss would eliminate every modeled case.
Read the 2024 Lancet Commission record | Read the UCL summary of the 2024 findings
A dose-response pattern in the Baltimore study
The Baltimore Longitudinal Study of Aging followed 639 adults for a median of 11.9 years. After adjustment for several demographic and health factors, the hazard of all-cause dementia increased by 27% for each 10-decibel worsening in hearing thresholds.
The severe-loss group contained only six participants and had a very wide confidence interval, so that estimate should not be treated as precise. The important finding was the overall graded pattern, not a promise that severity determines an individual outcome.
Newer cohorts add detail—not simple certainty
Framingham, 2025: In the incident-dementia sample of 935 adults age 60 and older, at least slight hearing loss was associated with a higher 15-year dementia risk (HR 1.71). The association was stronger among APOE ε4 carriers (HR 2.86). An overlapping MRI and cognitive sample included 1,656 participants and linked hearing loss with smaller brain volumes, more white-matter injury and executive-function decline.
Health ABC: Among 1,889 older adults, moderate or severe audiometric hearing impairment was associated with a 55% higher dementia hazard over nine years. Modeled continuously, each 10-decibel worsening was associated with a 14% higher hazard.
Southern Denmark: In 573,088 adults, hearing loss was associated with a modest 7% higher overall dementia hazard. Severe loss in the better ear and hearing loss without hearing-aid use were each associated with about a 20% higher hazard. The observational design cannot establish that hearing aids caused the difference.
Evidence across languages, countries and communities
The association is not limited to English-speaking populations. A systematic review of Sinitic tonal-language populations included 35 articles; 29 unique studies with approximately 372,154 participants entered the meta-analyses. Hearing loss was associated with incident dementia (pooled OR 1.89) and poorer cognitive performance, with no significant difference from findings in non-tonal-language populations.
Population-attributable fractions also vary because hearing-loss prevalence, access to care and other dementia risks differ by place. An international comparison reported hearing-loss PAF estimates of 3.9% in China, 6.4% in India and 7.7% in a Latin American sample. A New Zealand analysis estimated 6.5% for Māori and 8.0% for Europeans. These comparisons are useful for planning public-health priorities, but they are model-based estimates built from different surveys—not direct counts of dementia cases caused by hearing loss.
Sinitic-language systematic review | China, India and Latin America PAF analysis | New Zealand ethnicity-specific analysis
The practical conclusion: The public-health evidence is strong enough that hearing impairment belongs in serious dementia-risk discussions. The causal pathway is not fully settled, and risk estimates vary by method and population. Objective hearing testing, appropriate treatment, communication support and continued research are more defensible than either dismissing the connection or exaggerating hearing aids as a dementia cure.
Dementia is not the same thing as Alzheimer’s disease
Dementia is a broad term for changes in memory, reasoning, language or other thinking abilities that are serious enough to interfere with daily life. Alzheimer’s disease is the most common cause of dementia, but it is not the only cause. Vascular dementia, Lewy body dementia, frontotemporal dementia and mixed forms also occur.
Hearing loss is not dementia. A person who misses part of a question, answers incorrectly, withdraws from conversation or appears slow to respond may be struggling to hear rather than failing to understand. Hearing loss and dementia can also occur together, which makes careful communication and objective hearing testing especially important.
Important: A hearing evaluation does not diagnose dementia, and a hearing instrument specialist does not replace a neurologist, geriatrician, primary-care clinician or memory-care team. Hearing care addresses access to sound and communication—an essential piece of the larger picture.
What major studies have found
The studies below use different populations, hearing measures and definitions of cognitive decline. They should not be treated as identical. Together, however, they show why hearing loss is now taken seriously in brain-health research.
Hearing loss and incident dementia
Researchers followed 639 adults who were free of dementia at the start. Dementia risk increased as measured hearing loss became more severe. The study reported associations with both all-cause dementia and Alzheimer’s disease after adjustment for several other risk factors.
Objective hearing loss and nine-year dementia risk
In the Health, Aging and Body Composition study, moderate or severe audiometric hearing loss was associated with a higher risk of incident dementia over nine years compared with normal hearing. The researchers also examined changes in memory, perceptual speed and processing speed.
The ACHIEVE hearing-intervention trial
ACHIEVE enrolled 977 adults ages 70 to 84 with untreated hearing loss and no substantial cognitive impairment. Participants received either a best-practice hearing intervention or health education. In the total study population, the hearing intervention did not significantly reduce three-year cognitive decline. In a prespecified analysis of the older adults who entered through the higher-risk ARIC cohort, cognitive decline was about 48% slower in the hearing-intervention group.
Hearing loss, hearing-aid use and dementia in Denmark
This population-based cohort used hearing-test data and national health registries. Hearing loss was associated with a modestly higher dementia risk overall, with greater risk among people with more severe loss. The association was stronger among people with hearing loss who were not using hearing aids than among hearing-aid users. Because this was observational research, it cannot prove that hearing aids caused the difference.
Objective hearing tests found risk that self-report missed
In 2,946 older adults followed for up to eight years, researchers estimated that as much as 32% of incident dementia in that study population could be statistically attributable to audiometrically measured hearing loss. This is a population estimate, not a prediction for an individual and not proof that hearing loss caused those cases. Self-reported hearing difficulty did not capture the same association, reinforcing the value of objective testing.
Benefit varied with baseline cognitive risk
A later analysis applied a cognitive-risk model to ACHIEVE participants. Among participants in the highest quarter of predicted risk, three-year cognitive decline was 61.6% slower in the hearing-intervention group than in the health-education group. This helps explain why the original trial did not show the same effect across everyone and supports more individualized research.
How to read these numbers: A relative-risk percentage, hazard ratio or population-attributable fraction does not tell one person whether they will develop dementia. It describes patterns within a study population. Age, cardiovascular health, education, genetics, depression, physical activity, sleep, vision, social connection and many other factors also matter.
Researchers are studying several possible pathways
No single mechanism has been proven to explain the entire relationship. Several pathways may overlap.
1. Listening effort and cognitive load
When speech is unclear, the brain must spend more effort decoding sound. That can leave fewer mental resources available for remembering, reasoning and following a conversation.
2. Reduced auditory input and brain changes
Studies have connected poorer hearing with differences in brain structure, white-matter integrity and some biomarkers. Researchers are still determining whether these changes are causes, consequences or signs of shared aging processes.
3. Withdrawal and reduced engagement
Conversation becomes exhausting when words are repeatedly missed. Some people stop attending gatherings, calling friends or participating in activities. Social disengagement is itself associated with poorer health, although its exact role in the hearing-dementia pathway remains unsettled.
4. Shared health factors
Hearing loss and dementia can share underlying risks, including age and vascular disease. A common cause may contribute to both conditions without one directly causing the other.
5. Early cognitive change may affect hearing behavior
Early brain changes could make speech processing, device use or self-reporting more difficult before dementia is diagnosed. Researchers call this possible reverse causation.
6. More than one explanation may be true
The relationship may involve listening effort, brain stimulation, social participation, health conditions and early cognitive changes at the same time.
Review a large study of hearing, cognition, brain structure and tau-related findings
Hearing Loss, Cognition and Dementia: A Deeper Evidence Library
This library adds the long-term cohorts, randomized evidence, meta-analyses, brain imaging, dementia biomarkers, treatment studies and practical evidence for people already living with dementia. Study design matters: an association, a brain scan and a randomized intervention do not answer the same question.
How the evidence is built
ACHIEVE assigned hearing intervention or health education. The full sample result and the higher-risk subgroup result must both be reported.
Reviews combining dozens of cohorts consistently find an association between adult-onset hearing loss and later cognitive outcomes.
Hearing is measured before later decline or dementia, strengthening the timeline while leaving possible confounding.
MRI, tau and other measures test whether the association has biological signals beyond self-report and cognitive scores.
Communication, quality of life, caregiver support, device management and hearing-access accommodations matter even without proven dementia prevention.
Pooled hazard ratio for incident dementia in a 2024 review of 50 cohorts and more than 1.5 million participants.
Increase in pooled dementia risk for each 10-decibel worsening of hearing in that review.
Slower decline in the higher-risk ARIC subgroup of the ACHIEVE randomized trial.
Slower decline in ACHIEVE participants in the highest quarter of predicted cognitive risk.
Do not combine these percentages. They use different populations, outcomes and statistical measures. None is a personal prediction.
Evidence that adds more than another headline
2013Faster cognitive decline with measured hearing loss
In 1,984 Health ABC participants, hearing loss was associated with annual decline that was 41% greater on a global cognitive test and 32% greater on an executive-function test. Over six years, it was associated with a 24% higher risk of incident cognitive impairment. This was observational evidence.
2014Longitudinal MRI and faster brain-volume loss
Repeated MRI scans in 126 adults over an average of 6.4 years linked hearing impairment with faster whole-brain volume loss and greater loss in right temporal regions involved in sound, language and memory.
2018Memory trajectory before and after hearing-aid adoption
Among 2,040 adults followed for 18 years, episodic-memory decline was slower after people began using hearing aids. Users can differ from nonusers in health, resources and access to care, so the device cannot be isolated as the cause.
2019Loneliness and isolation explained part of the association
An 8,199-person English cohort found partial mediation through loneliness and social isolation. Other studies found little mediation, supporting social withdrawal as one pathway rather than the entire explanation.
2020Hippocampal and white-matter differences
In 1,086 dementia-free older adults, hearing loss was associated with lower integrity in temporal and limbic white-matter pathways and lower gray-matter integrity in the hippocampus.
2021Speech-in-noise performance predicted dementia
Among 82,039 UK Biobank participants followed for about 11 years, insufficient speech-in-noise performance was associated with a 61% higher dementia risk and poor performance with a 91% higher risk. Depression and isolation explained little of the association.
2023Review of hearing aids and cochlear implants
A review of 31 studies and 137,484 participants found hearing-restorative device use was associated with a 19% lower hazard of long-term cognitive decline and a small short-term cognitive-score improvement. Most long-term evidence was observational.
2024Fifty cohorts and more than 1.5 million participants
Adult-onset hearing loss was associated with incident dementia, mild cognitive impairment, cognitive decline and Alzheimer’s disease dementia. The pooled dementia hazard ratio was 1.35; vascular-dementia findings were not conclusive.
2024Lancet dementia-prevention framework
The 2024 Lancet Commission retained hearing loss among 14 potentially modifiable factors and estimated the combined factors could theoretically account for about 45% of dementia cases at a population level. That is not a guarantee for an individual and is not a hearing-aid treatment effect.
2025Tau, amyloid and brain-atrophy review
Across 66 studies, adult-onset hearing loss was more consistently associated with temporal-region atrophy and elevated tau than with beta-amyloid. Twenty-five of 44 cross-sectional and six of nine longitudinal analyses found temporal atrophy associations; five of six analyses found elevated in-vivo tau, compared with four of 17 for amyloid.
2025Hearing plus vision impairment
A meta-analysis of 11 studies and 346,659 people found combined hearing and vision impairment was associated with a 46% higher dementia hazard and a stronger association with Alzheimer’s disease. Sensory care should include vision as well as hearing.
2025Framingham brain structure and 15-year risk
At least slight hearing loss was associated with more white-matter hyperintensity volume and higher all-cause dementia risk. Mild or greater loss was associated with smaller brain volumes and executive-function decline, with a stronger association among APOE ε4 carriers.
2026Effective improvement mattered more than device ownership
A pooled analysis of 61,089 hearing-impaired adults in 33 countries associated hearing-aid use with a 9% lower probable-dementia hazard. Users reporting good hearing improvement had a 14% lower hazard; poor improvement showed no significant reduction. It remains observational evidence.
Several pathways may operate together
The strongest interpretation: hearing loss may contribute to decline for some people, may share causes with dementia, and may sometimes be an early marker of brain change. The research does not support the simple statement that hearing loss directly causes every case of Alzheimer’s disease.
What hearing treatment may change
| Evidence | Finding | Limit |
|---|---|---|
| ACHIEVE trial | No significant benefit across the full sample; about 48% slower decline in the prespecified higher-risk ARIC group. | A subgroup finding is not a universal hearing-aid effect. |
| ACHIEVE risk analysis | 61.6% slower decline in the highest quarter of predicted cognitive risk. | Secondary analysis requiring confirmation. |
| 2023 device review | 19% lower long-term cognitive-decline hazard associated with hearing devices. | Most long-term evidence was observational. |
| 2026 pooled study | Lower probable-dementia risk only among users reporting effective hearing improvement. | Effectiveness and outcomes were not randomized. |
A widely shared 2023 hearing-aid study was retracted
A UK Biobank paper reported that hearing aids removed the additional dementia risk associated with hearing loss. The Lancet Public Health retracted it after the authors discovered that data for users and nonusers had been switched, rendering the findings false and misleading. This page does not use that study as evidence. Read the official retraction.
Communication and quality of life become the immediate goals
For families deciding whether a device can be managed day to day, see our practical guide to hearing aids for seniors with memory loss or dementia.
Estimated prevalence of measured hearing loss among U.S. adults living with dementia.
Reported hearing-aid use among participants with both dementia and hearing loss.
Met criteria for a 2024 review of hearing-aid treatment in people with dementia.
Improved in that review; cognitive outcomes did not show a clear effect.
Hearing loss is frequently missed or undertreated
The nationally representative study found hearing loss in nearly four out of five people living with dementia and in more than nine out of ten age 85 or older.
Device success requires a care system
Memory changes can affect charging, insertion, storage and recognizing the device. A simple routine, labeled storage, caregiver checks, automatic settings and regular maintenance may be as important as the model selected.
Quality of life evidence is stronger than cognition evidence
The dementia-specific treatment review found only five eligible clinical studies. Hearing aids appeared to improve quality of life, but cognitive outcomes did not show a clear change.
Hearing-adapted cognitive testing matters
The MoCA-H uses written presentation and substituted items to reduce auditory bias. Standard spoken tests can underestimate performance when words or instructions are missed.
From the ear to the brain


Journey of Sound to the Brain. NIH/NIDCD public-domain animation showing how sound moves from the ear to the brain and is interpreted.
Questions the science has not answered
Who receives cognitive benefit?
Higher-risk groups may benefit more, but researchers need reliable clinical markers that do not turn subgroup findings into promises.
How early should treatment begin?
Evidence is strongest in older adults. Midlife timing, duration of untreated loss and long-term effective use need better trials.
Does fitting quality change outcomes?
Verification, speech-in-noise benefit, counseling, follow-up and daily use may matter more than simply owning a device.
Cause, shared disease or early marker?
The balance among these explanations may differ by person, hearing-loss type, dementia subtype and vascular or genetic risk.
ACHIEVE Brain Health Follow-Up Study
The NIH-sponsored follow-up is tracking 629 original participants for long-term cognitive, dementia and brain outcomes. ClinicalTrials.gov listed it as active and not recruiting in June 2026, with completion estimated for May 2027. View the official trial record.
Research standard used on this page: randomized results are reported with their full-sample findings; observational studies are labeled observational; population estimates are not presented as individual predictions; and retracted work is identified rather than quietly repeated.
The bottom line in plain English
First: hearing loss does not mean a person will develop dementia. It is one risk factor among many, and most studies describe averages across large groups.
Second: the relationship is too consistent to dismiss. Objective hearing loss, speech-in-noise difficulty and more severe loss repeatedly appear alongside higher rates of cognitive decline or dementia.
Third: science has not settled on one explanation. Extra listening effort, reduced auditory input, isolation, vascular disease and early brain changes may all play a part.
Fourth: hearing treatment is worthwhile without making a dementia-prevention promise. Clearer communication, participation, safety and reduced frustration are meaningful outcomes now.
Finally: when a person already has memory loss or dementia, hearing care should become more patient and practical—not less important. The plan must respect the person, involve trusted support when appropriate and use technology that can actually be managed.
Hearing difficulty can look like memory difficulty
A person may appear confused when the real problem is that part of the message never arrived clearly. This can happen at home, in a doctor’s office or during a cognitive screening.
- Answering a different question from the one that was asked
- Taking longer to respond because the person is reconstructing missed words
- Forgetting instructions that were never heard accurately
- Withdrawing, nodding or saying “yes” to avoid repeated embarrassment
- Becoming irritable or fatigued after sustained listening effort
Before assuming cognitive decline
- Reduce background noise and face the person.
- Confirm that hearing aids are charged, working and inserted correctly.
- Speak clearly at a normal pace; do not shout.
- Rephrase rather than repeating the same unclear sentence louder.
- Ask the person to explain instructions in their own words.
- Request hearing-access accommodations during cognitive testing when needed.
Researchers have developed hearing-adapted cognitive tools because standard spoken tests can be affected by hearing impairment.
Read about the validated MoCA-H for people with hearing impairment
Clear expectations protect patients
Hearing care can help with
- Access to speech and important environmental sounds
- Participation in family conversations and medical appointments
- Reducing avoidable listening strain
- Communication between patients, families and caregivers
- Safety cues, routines and daily independence
- Finding technology that is realistic for the person’s abilities and support system
Hearing care cannot promise
- Prevention of dementia or Alzheimer’s disease
- Reversal of established cognitive disease
- A medical diagnosis of memory loss
- That every patient will tolerate or manage the same device
- That amplification alone will fix every communication problem
A hearing aid is not a dementia treatment. It is a communication tool. That distinction matters. Better access to sound can still be deeply valuable when a person already has mild cognitive impairment, dementia or Alzheimer’s disease.

Gwen Cantrell, BC-HIS, CDP
Gwen’s Certified Dementia Practitioner credential reflects a deliberate commitment to continuing education—not a marketing phrase. She pursued specialized dementia-care education because hearing loss, memory changes and caregiver communication often meet in the same appointment.
Through the National Council of Certified Dementia Practitioners, Gwen completed the Alzheimer’s Disease and Dementia Care seminar and the separate CDP certification process. NCCDP’s curriculum covers person-centered care, compassionate communication, behavioral expressions, dementia-friendly environments, fall prevention, activities of daily living, depression, sleep changes, delusions, hallucinations, intimacy, spirituality and end-of-life care. The credential is renewed every two years with continuing-education requirements.
That training strengthens Gwen’s ability to recognize when a standard hearing appointment needs to slow down, involve a trusted family member, use simpler instructions or choose a device plan that will actually work in the patient’s daily life.
See Gwen’s full background and credential details | Official NCCDP certification requirements
Make hearing support part of the care plan
- Request objective hearing testing. People often underestimate their own hearing difficulty.
- Bring the hearing devices. Include the charger, batteries, cleaning tools, remote control or phone used with the devices.
- Describe the real routine. Who helps in the morning? Who charges the devices? Where are they stored?
- Report specific situations. “She cannot follow dinner conversation” is more useful than “her hearing is bad.”
- Watch for sudden change. Rapid confusion, abrupt behavior change or sudden hearing loss requires medical attention rather than a routine hearing-aid adjustment.
- Keep expectations humane. Consistent use may take repetition, cueing and caregiver support. Blame does not improve adaptation.
Daily communication habits
- Get the person’s attention before speaking.
- Stay in the same room and keep your face visible.
- Turn off television or competing sound.
- Use short, complete sentences—not childish language.
- Give one instruction at a time.
- Pause long enough for a response.
- Rephrase when a message is missed.
- Confirm important dates and instructions in writing.
- Check device function before assuming the person is refusing to listen.
Frequently asked questions
Does hearing loss cause dementia?
Research consistently finds an association, but association alone does not prove direct causation. Shared health factors, brain changes, listening effort, social withdrawal and reverse causation may all contribute. It is more accurate to say hearing loss is a potentially modifiable risk factor being actively studied.
Can hearing aids prevent Alzheimer’s disease?
No clinician can responsibly promise that. The ACHIEVE trial did not find an overall cognitive benefit across every participant. It did find slower cognitive decline in higher-risk groups, which is encouraging and deserves further study. Hearing aids should be recommended for hearing and communication needs, not sold as an Alzheimer’s cure.
Should a person with dementia still have a hearing test?
Usually, yes. Hearing loss can worsen communication and may be missed when every difficulty is attributed to dementia. The test and appointment may need more time, visual cues, caregiver input and a quieter setting.
Can dementia make hearing aids harder to use?
Yes. Memory changes may affect charging, insertion, cleaning, recognizing the device or remembering why it is needed. Simpler controls, clear routines, labeled storage and caregiver support can help.
Can hearing loss affect cognitive screening results?
It can. Many cognitive tests depend on hearing spoken instructions and words. Missed speech can lower performance even when the underlying problem is hearing access. The medical team should know about the hearing loss and consider appropriate accommodations or hearing-adapted tools.
What if the person says their hearing is fine?
Self-report often underestimates age-related hearing loss. The 2025 ARIC-NCS study found that objective audiometric hearing loss captured an association with dementia that self-report did not. A professional hearing evaluation provides more useful information than guessing.
Does Gwen diagnose dementia?
No. Gwen provides hearing evaluation, hearing-aid fitting and communication support within her professional hearing-care role. Her CDP education helps her work more effectively and respectfully with patients living with dementia and the people who care for them.
Research and patient resources
These links go to the original studies, PubMed records or an official National Institute on Aging patient resource.
Prospective study examining measured hearing loss and later all-cause dementia and Alzheimer’s disease.
Nine-year analysis of audiometric hearing impairment, dementia and cognitive change.
Best-practice hearing intervention versus health education over three years.
Large population-based cohort using hearing examinations and national health registries.
Open-access study comparing objective hearing tests with self-reported hearing.
Secondary ACHIEVE analysis showing that treatment effects varied by predicted cognitive risk.
Research examining possible biological pathways across several large datasets.
A hearing-adapted cognitive screening tool designed to reduce auditory bias.
Patient-friendly information on hearing loss, communication, cognitive health and treatment.
Official description of Gwen’s CDP credential and professional-development expectations.
Reviewed and contributed to by Gwen Cantrell, BC-HIS, CDP
Board Certified Hearing Instrument Specialist • Georgia-licensed Hearing Aid Dispenser
